Fig. 3

A H&E staining in intestinal tissue: a sham group, b II/R model group, c 40 mg/kg Emodin treatment group, d TCN treatment before II/R, e Treatment with 40 mg/kg of Emodin following TCN treatment before II/R, f SNPP treatment before II/R, g Treatment with 40 mg/kg of Emodin following SNPP treatment before II/R. B Akt and HO-1 inhibitor administration effectively counteracted the protective effects of Emodin in mitigating II/R injury and promoted the occurrence of apoptosis. Emodin represents II/R plus 40 mg/kg Emodin. Quantitative analysis of the expression compared with β-actin, as shown alongside